If you have no arguments and want to hide information, the easiest way is to block the person

More good news -

I have just been blocked by Frances Leader for commenting on nanotechnology.

She blocked me and deleted (hid?) all of her rather rude correspondence with me (and my replies).

This is what the movement of people pretending to be “freedom fighters” really looks like...

It would be laughable if it weren't for the fact that people are actually dying as a result of the toxicity of this technology, and that is tragic.

From this discussion:

Here is the study:

https://healthnewsdigest.com/2021/03/23/chop-researchers-find-supplement-prevents-strokes-in-patients-with-rare-genetic-disorder/

Since the formation of amyloid-producing proteins is already implicated in HCCAA and other diseases, researchers have studied whether drugs that reduce the aggregation of these proteins have the potential to reduce the toxic oligomers, or polymers made up of a few different molecules. To properly study this strategy in HCCAA, the research team created cell lines expressing both wild type and L68Q-mutant hCC and then attempted to non-toxically interfere with the aggregation of the amyloid-producing proteins. The researchers also studied a supplement called N-acetyl-cysteine (NAC), which is sometimes prescribed to break up mucous in the lung and has also been shown to protect against the toxic liver damage caused by an acetaminophen overdose, to determine whether it had an effect on hCC-amyloid protein deposits in skin biopsies of patients with a known diagnosis of HCCAA.

The researchers found that treating these cell lines with NAC breaks the oligomers into monomers, or molecules that have been separated from the chain that brings them together. This in turn helps to prevent the formation of amyloid-producing proteins that lead to the amyloid deposits implicated in strokes and other impairments. The researchers also performed skin biopsies on six patients with the L68Q-hCC variant taking NAC to determine levels of hCC-amyloid protein deposits following treatment. Five of the six patients saw between a 50% and 90% reduction of L68Q-hCC levels, suggesting that this variant is a clinical target for reducing agents such as NAC. This proof-of-concept study led to a clinical trial to see if these results are observed in a larger patient cohort.

“Amyloids cannot precipitate without aggregating, so if we can prevent that aggregation with a drug that is already available, then we could make an incredible difference in the lives of these patients,” Hakonarson said. “Additionally, since we already have genetic testing available to identify these patients, we could conceivably give this treatment early in life and potentially prevent that first stroke from ever occurring.”

Trust the science...

https://web.archive.org/web/20071024185454/http://aliveandwellsf.org/articles/derosa_NAC_GSH_2000.pdf

Dosing of NAC

downloads.hindawi.com/journals/jnme/2021/9949453.pdf

8.9. Dosing of NAC. N-acetylcysteine dosing varies significantly with various clinical studies and doses of 1200 mg daily or more are usually required to be clinically relevant. Studies in metabolic diseases show that 5–600 mg orally per day may be sufficient to ameliorate fatty liver disease. For Crohn’s disease and ulcerative colitis, doses of 800 mg perday seemed sufficient. Doses as high as 1250 mg orally three times a day have been used safely in MS and showed benefit in reducing fatigue. Doses of 8000 mg/day orally did not cause clinically significant reactions in HIV patients [150]. Clearly dosing is still debated and much needs to be learned

Have a great day!