ROBERT MALONE AND COUNTERMEASURES AGAINST ZIKA
Robert Malone developed Zika vaccine:
https://loop.frontiersin.org/people/1131095/bio Robert Wallace Malone Brief Bio
Ebola vaccine project for NewLink/Bioprotection Systems (rVSVdG ZEBOV Ebola vaccine project), resulting in well over 100M USD non-dilutive capital to NL/BPS. This also included working with the World Health Organization as well as initial set up of the licensing deal to Merck Vaccines of the Ebola vaccine.
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4999274/ Zika Fetal Neuropathogenesis: Etiology of a Viral Syndrome - PMC (nih.gov)
Zachary A. Klase, Svetlana Khakhina, Adriano De Bernardi Schneider, Michael V. Callahan, Jill Glasspool-Malone, and Robert Malone
Abstract
The ongoing Zika virus epidemic in the Americas and the observed association with both fetal abnormalities (primary microcephaly) and adult autoimmune pathology (Guillain–Barré syndrome) has brought attention to this neglected pathogen. While initial case studies generated significant interest in the Zika virus outbreak, larger prospective epidemiology and basic virology studies examining the mechanisms of Zika viral infection and associated pathophysiology are only now starting to be published. In this review, we analyze Zika fetal neuropathogenesis from a comparative pathology perspective, using the historic metaphor of “TORCH” viral pathogenesis to provide context. By drawing parallels to other viral infections of the fetus, we identify common themes and mechanisms that may illuminate the observed pathology. The existing data on the susceptibility of various cells to both Zika and other flavivirus infections are summarized. Finally, we highlight relevant aspects of the known molecular mechanisms of flavivirus replication.
Unanswered Questions
In order to more completely understand the link between ZIKV infection and fetal abnormalities, more work must be done. The characteristic presentation of Zika congenital syndrome ranges from viral centric (microcephaly, blindness, ventricular calcifications, and fetal presence of ZIKV by rt-PCR) to another extreme (long bone dysgenesis, negative for ZIKV) possibly associated with placental insufficiency.
The gaps in understanding of ZIKV neuropathology highlighted in this review suggest that efforts should first be focused on obtaining clear, statistically significant data addressing a few specific questions. Prospective case control study reports on ZIKV infection of pregnant women and fetal outcomes are a step in the right direction. As such studies continue, a more definitive correlation between ZIKV infection and various congenital outcomes will become possible. Additionally, fundamental research will be required to answer questions regarding the ability of ZIKV to cross the placenta and infect the developing brain.
COULD THE VACCINE HAVE CAUSED THESE AFOREMENTIONED INJURIES?
https://apnews.com/article/18d0a9394e8b4b46a10cef5ceb59d59e No proof Zika causes microcephaly, UW-Madison study says
David Wahlberg | Wisconsin State JournalDecember 27, 2017
There isn’t enough evidence to claim that Zika virus causes abnormally small heads in babies of infected mothers, according to a UW-Madison study that challenges government findings.
A government official said evidence of the link is “sufficient and growing.”
An increase in reported Zika infections during pregnancy was not followed by an increase in cases of microcephaly, said Bautista, a UW-Madison associate professor of population health sciences.
In addition, he found, babies of Zika-infected mothers didn’t have significantly more microcephaly, which can cause developmental problems and even death.
One factor that made it seem like there was a spike in microcephaly cases in 2015 is that criteria for the condition changed that year to include a larger head size, he said. Studies suggesting a link between Zika and microcephaly were too small to draw conclusions and didn’t account for genetic causes of the birth defect, he said.
The Centers for Disease Control and Prevention and the World Health Organization, which said pregnant women should avoid traveling to Brazil and other places affected by Zika, declared in April 2016 that the virus caused microcephaly.
The CDC published its findings in the New England Journal of Medicine, saying its scientists had conducted a “rigorous weighing of evidence.”
Other researchers have questioned the link between Zika, which is mostly spread by mosquitoes, and microcephaly. But Bautista said his study is the first one published to analyze the evidence and call it into question.
The CDC and other government agencies “jumped to the conclusion that these two events were associated,” he said Wednesday in an interview.
In a public talk about his research earlier this year, Bautisa said
the agencies “created a false crisis.”
“It points to a major failure of the peer review system in health sciences,” he said. “It sets a dangerous precedent by establishing causal links mostly be official decree.”
https://videos.med.wisc.edu/videos/72293 Zika: Innocent Until Proven Guilty - SMPH Video Library (wisc.edu)
https://www.sciencedirect.com/science/article/abs/pii/S1047279717302831 Maternal Zika virus infection and newborn microcephaly—an analysis of the epidemiological evidence - ScienceDirect
Abstract
Purpose
To evaluate whether existing data and evidence support a causal link between maternal Zika virus (ZIKV) infection and newborn microcephaly.
Methods
I quantified and compared the prevalence of all and severe microcephaly in Brazil, during and before 2015–2016, to assess whether an outbreak has occurred, used time series analysis to evaluate if the presumed outbreak was linked to a previous outbreak of ZIKV infections, and quantitatively synthesized published data from observational studies testing this association.
Results
The prevalences of microcephaly in 2015–2016 were similar or lower than background levels (prevalence ratio [PR] for all microcephaly: 0.19; 95% confidence intervals [CI]: 0.17, 0.20). Changes in the number of cases of ZIKV infections at times matching 11–18 weeks of pregnancy were not followed by changes in the number of microcephaly cases (PR for infection at 12 weeks: 1.02; 95% CI: 0.99, 1.05). In observational studies, the prevalence of microcephaly was not significantly increased in newborns of Zika-infected mothers (average PR: 1.30; 95% CI: 0.84, 2.02).
Conclusions
Existing evidence is insufficient to claim maternal ZIKV infection causes microcephaly. Although a public health response seems sensible, it should be consistent with existing knowledge and consider risks, potential benefits and harm, and competing priorities.
But:
https://www.sciencedirect.com/science/article/abs/pii/S1071909107000496 Microcephaly Syndromes - ScienceDirect
Microcephaly can be due to either genetic or environmental causes.
https://www.tandfonline.com/doi/full/10.1016/S0968-8080%2807%2930333-9 Full article: Research on Severe Maternal Morbidities and Near-Misses in Brazil: What We Have Learned (tandfonline.com)
Using this same approach, we recently evaluated the situation in Brazil by geographical region from data in a 1996 demographic and health survey. This showed a varied panorama of reported complications, ranging from around 15% in the state of São Paulo to as high as 23% in the northern part of the country. These variations were in line with the degree of economic and human development of the different regions of the country, with complications being significantly greater in the east central, northeast, north and west central parts of the country, the least developed areas of Brazil.
Some researchers, including Robert W Malone, look for a correlation between Zika virus infection and congenital brain abnormalities and Guillain-Barré syndrome.
https://dash.harvard.edu/bitstream/handle/1/29407704/4999274.pdf?sequence=1 Zika Fetal Neuropathogenesis: Etiology of a Viral Syndrome (harvard.edu)
The ongoing Zika virus epidemic in the Americas and the observed association with both fetal abnormalities (primary microcephaly) and adult autoimmune pathology (Guillain–Barré syndrome) has brought attention to this neglected pathogen.
https://onlinelibrary.wiley.com/doi/10.1111/cla.12178 Molecular evolution of Zika virus as it crossed the Pacific to the Americas - Bernardi Schneider - 2017 - Cladistics - Wiley Online Library
Abstract
Zika virus was previously considered to cause only a benign infection in humans. Studies of recent outbreaks of Zika virus in the Pacific, South America, Mexico and the Caribbean have associated the virus with severe neuropathology. Viral evolution may be one factor contributing to an apparent change in Zika disease as it spread from Southeast Asia across the Pacific to the Americas. To address this possibility, we have employed computational tools to compare the phylogeny, geography, immunology and RNA structure of Zika virus isolates from Africa, Asia, the Pacific and the Americas.
These findings support the working hypothesis that mutations acquired by Zika virus in the Pacific and Americas contribute to changes in pathology.
A more severe Zika disease spectrum (Guillain-Barré Syndrome (GBS; autoimmune disease that attacks the nervous system often postinfection) and fetal microcephaly) was observed in both the 2013–2014 outbreak in French Polynesia and in the Americas (Oehler et al., 2014; ECDC, Stockholm, 2016).
Much remains to be understood about Zika virus, as previously it was associated only with mild disease and thus understudied.
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4774925/ Zika Virus: Medical Countermeasure Development Challenges - PMC (nih.gov)
Reports of high rates of primary microcephaly and Guillain–Barré syndrome associated with Zika virus infection in French Polynesia and Brazil have raised concerns that the virus circulating in these regions is a rapidly developing neuropathic, teratogenic, emerging infectious public health threat. There are no licensed medical countermeasures (vaccines, therapies or preventive drugs) available for Zika virus infection and disease.
Zika virus infection has spread rapidly in the tropical Americas since introduction to Brazil in 2014. Although a causal association is not yet confirmed, there is a growing consensus that Zika infection is linked to an upsurge in cases of Guillan Barré (GBS) syndrome and the birth of microcephalic infants following maternal infection.
https://www.biorxiv.org/content/10.1101/044834v1.full.pdf Antibody mediated epitope mimicry in the pathogenesis of Zika virus related disease
We hypothesized that antibodies to Zika virus epitopes may contribute to the pathogenesis of Guillain Barré syndrome, microcephaly and ocular lesions associated with Zika virus, and may be a driver of autoimmunity.
These studies are not conclusive, of course. Despite this, pregnant women in Brazil have been injected with experimental vaccines.
The Ebolavirus vaccine (rVSVΔG-ZEBOV-GP) is stored at -80 to -60 °C and should be kept frozen for transport.
https://www.aha.org/system/files/media/file/2021/03/infographic-vaccine-storage.pdf
COVID-19 Vaccine is stored at -80 to -60 °C and should be kept frozen for transport.
[Prior to thawing, store in an ultra-cold freezer between -80°C to -60°C]
Is this the same technology?
So, what about possible adverse effects of these injections?
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8103672/pdf/main.pdf Evaluation of Covid-19 vaccines: Pharmacoepidemiological aspects
Pharmacoepidemiological strategies and planned activities for SARS-CoV-2 vaccine monitoring
The list of these adverse events of specific interests has been defined by the Safety Platform for Emergency vACcines (SPEAC) and the Brighton Collaboration, and was completed in Europe with additional events. It initially included
enhanced disease (!!!!!)
following immunization, multisystem inflammatory syndrome in children, acute respiratory distress syndrome, acute cardiovascular injury, coagulation disorder, acute kidney injury, acute liver injury, generalized convulsion,
Guillain-Barré syndrome (!!!!!),
meningoencephalitis, acute disseminated encephalomyelitis, anosmia and dysgueusia, chilblain–like lesions, single organ cutaneous vasculitis, multiform erythema, anaphylaxis, acute aseptic arthritis, thrombocytopenia. It was completed in Europe with narcolepsy, transverse myelitis and events relating to pregnancy outcome (preterm birth, major congenital anomalies,
microcephaly (!!!!!),
fetal growth restriction, gestational diabetes, preeclampsia, termination of pregnancy for fetal anomaly, neonatal death, and spontaneous abortions and stillbirth).
Oxidative stress, microcephaly and Guillain-Barré syndrome - toxicity of vaccine components and/or presence of environmental toxins.
https://www.mdpi.com/1422-0067/24/4/4242 IJMS | Free Full-Text | Neuroinflammation and Oxidative Stress in Individuals Affected by DiGeorge Syndrome (mdpi.com)
3. Oxidative Stress in DiGeorge Syndrome
Oxidative stress plays an important role in the development of different signs and symptoms of various genetic syndromes. The correlation between the imbalance of the redox state and the clinical DGS manifestations has not yet been established, but important findings have been revealed, as shown in Table 1. In particular, in 2019, Fernandez et al. used a mouse model of DiGeorge/22q11 deletion syndrome to describe biological mechanisms of neuronal under-connectivity, which reflects reduced growth of dendrite, axon and synapse. It seems that connections between and within regions of the cerebral cortex are the basis for complex behaviors. It has been proposed that the under-connectivity of association cortices underlies behavioral deficits in several neurodevelopmental disorders, including DGS.
In conclusion, it is still not possible to describe a direct connection between oxidative stress and all the clinical manifestations of patients with 22q11DS deletion, but recent studies have pointed out that there could be a link between the psychiatric manifestations and alteration of crucial genes for the mitochondrial metabolism, leading to a reduction of antioxidant defenses. Further studies are needed to evaluate this connection and eventually the benefit of an antioxidant therapy.
Conclusions
Based on the available information, it emerges that some of the clinical manifestations of patients with DGS are directly related to alterations both in the mitochondrial mechanisms that regulate oxidative stress and in the mechanisms of neuroinflammation.
As for oxidative stress, there are no studies explaining the clinical relationship between DGS and oxidative stress, but there are interesting preliminary studies about the molecular and biological alterations related to them. In fact, the loss of the two genes DGCR8 and TXNRD2 may determine an increase in ROS production and, at the same time, antioxidant depletion.
In DGS mouse models showing alterations in mitochondrial function, it has been observed that a loss of projection neurons in layers 2 and 3 of the cortex is associated with significant cognitive impairments. Furthermore, it emerges that neuroinflammation is directly linked to the development of psychiatric symptoms, especially psychotic, anxious and those related to ASD. In fact, it is clear from the literature that the increase in proinflammatory cytokines, such as IL-1, IL-6 and TNF-α, and imbalances in T cells are directly responsible for the development of psychiatric disorders.
At the same time, neuroinflammation would alter the mechanisms underlying the correct development of the brain and synaptic plasticity, confirming its importance also in the development of cognitive disorders. Thus, the use of antioxidants such as the polyphenols extracted from plants, possessing also anti-inflammatory properties, could be a useful tool to counteract the deleterious DGS effects on the affected individuals as previously shown in humans and animals models for other pathologies.
https://pubmed.ncbi.nlm.nih.gov/17179587/ [The role of oxidative stress in pathogenesis of GBS] - PubMed (nih.gov)
We aimed to determine the role of oxidative metabolism in viral polyneuropathy pathogenesis. The activity of pro- and antioxidant systems of the body was studied by electron paramagnetic resonance (EPR) method. In blood and cerebrospinal fluid the intensive EPR signals of nitric oxide (NO), complexes of NO with nonhemic iron (HbNO), lypo- and superoxide radicals content noticeably increases, the signals of free Mn2+ and Fe2+ revealed, the activity of blood antioxidant enzymes, ceruloplasmin and katalasa increases (by 60%), superoxidedismitase's and glutation reductases activity decreases (by 20% and 70% correspondingly). It was considered, that inflammatory damage of nervous system induced by different infectious stimulus is initiated by activated immune cell proinflamatory agents (reactive oxygen and nitrogen species). Subsequently the oxidative stress, as result of accumulation of generators of reactive oxygen species, disordered intracellular metabolism products, contributes to axon demielinization and degeneration.
https://www.youtube.com/watch?v=Qv9cd07ToX0 Oxidative stress in Guillain Barre Syndrome - YouTube
https://www.sciencedirect.com/science/article/pii/B9780128202678000121 Oxidative stress in Guillain-Barré syndrome and linkage with neurology - ScienceDirect
To quote Jon Rappoport:
https://blog.nomorefakenews.com/2021/03/04/zika-was-a-warm-up-for-covid-it-didnt-fly/ Zika was a warm-up for COVID; it didn’t fly « Jon Rappoport's Blog (nomorefakenews.com)
Zika was a warm-up for COVID; it didn’t fly
I covered the Zika outbreak extensively in 2016. It was yet another fraud, and it collapsed under the weight of warnings to women to avoid pregnancy. Women wouldn’t obey in great enough numbers.
Basically, the official position was: an outbreak of microcephaly was occurring, worldwide, starting in Brazil. Babies were being born with smaller heads and brain damage. The cause was the Zika virus, carried by mosquitoes.
When I was exposing the lies, in 2016, I wasn’t questioning the existence of the Zika virus. Now, in 2021, I would be demanding proof that the virus had actually been isolated.
Here are excerpts from the many articles I wrote during the “Zika crisis”. There is more, much more to the story, but what I’m publishing here is enough to reveal the standard pattern of pandemic ops: pretend the “medical condition” is entirely the result of a germ; fake the exact cause; cover up ongoing government/corporate crimes.
EXCERPT ONE, 2016: There is no convincing evidence the Zika virus causes the birth defect called microcephaly.
Basically, Brazilian researchers, in the heart of the purported “microcephaly epidemic,” decided to stop their own investigation and simply assert Zika was the culprit. At that point, they claimed that, out of 854 cases of microcephaly, only 97 showed “some relationship” to Zika.
You need to understand that these figures actually show evidence AGAINST the Zika virus as the cause. When researchers are trying to find the cause of a condition, they should be able to establish, as a first step, that the cause is present in all cases (or certainly an overwhelming percentage).
This never happened. The correlation between the presence of Zika virus and microcephaly was very, very weak.
As a second vital step, researchers should be able to show that the causative virus is, in every case, present in large amounts in the body. Otherwise, there is not enough of it to create harm. MERE PRESENCE OF THE VIRUS IS NOT ENOUGH. With Zika, proof it was present in microcephaly-babies in large amounts has never been established.
But researchers pressed on. A touted study in the New England Journal of Medicine claimed Zika infected brain cells in the lab. IRRELEVANT. Cells in labs are not human beings. The study also stated that Zika infected baby mice. IRRELEVANT. Mice are not humans. And these mice in the lab had been specially altered or bred to be “vulnerable to Zika.” USELESS AND IRRELEVANT.
EXCERPT TWO, 2016: Millions of bees have just died in South Carolina, because Dorchester County officials decided to attack Zika mosquitoes from the air, from planes, with a pesticide called Naled.
The Washington Post reports, in an article headlined: “‘Like it’s been nuked’: Millions of bees dead after South Carolina sprays for Zika mosquitoes.”
“The county acknowledged the bee deaths Tuesday. ‘Dorchester County is aware that some beekeepers in the area that was sprayed on Sunday lost their beehives,’ Jason Ward, county administrator, said in a news release. He added, according to the Charleston Post and Courier, ‘I am not pleased that so many bees were killed.’”
That’s the highest degree of outrage County Administrator Ward can muster? He’s not pleased?
If you want to dig further, you can discover that, despite assurances to the contrary, Naled, like other toxic organophosphate pesticides, harms humans as well. Organophosphates are neurotoxins. The original research was done in Germany, in the hunt for nerve-agent weapons.
And how about this? The cure for the problem causes the problem…
Naled, the organophosphate pesticide now being sprayed on Miami to kill “Zika mosquitoes,” has dire effects.
Reference: a 2014 study, “Neurodevelopmental disorders and prenatal residential proximity to agricultural pesticides: the CHARGE study.” [Environmental Health Perspectives, 2014 Oct;122(10):1103-9.]
Key quotes from the study:
“Gestational exposure to several common agricultural pesticides can induce developmental neurotoxicity in humans, and has been associated with developmental delay and autism.” [Emphasis added]
“We evaluated whether residential proximity to agricultural pesticides during pregnancy is associated with autism spectrum disorders (ASD) or developmental delay (DD)…”
“Approximately one-third of CHARGE study mothers lived, during pregnancy, within 1.5 km (just under 1 mile) of an agricultural pesticide application. Proximity to organophosphates at some point during gestation was associated with a 60% increased risk for ASD [Autism Spectrum Disorders], higher for third-trimester exposures…and second-trimester chlorpyrifos [an organophosphate pesticide] applications…”
“This study of ASD strengthens the evidence linking neurodevelopmental disorders with gestational pesticide exposures, particularly organophosphates…”
The pesticide spraying affects pregnant mothers by raising the risk of neurological damage to their babies.
EXCERPT THREE: Here’s an “oops” Zika revelation:
“New doubts on Zika as cause of microcephaly.” ScienceDaily, 24 June 2016.
Source: New England Complex Systems Institute
“Brazil’s microcephaly epidemic continues to pose a mystery — if Zika is the culprit, why are there no similar epidemics in other countries also hit hard by the virus? In Brazil, the microcephaly rate soared with more than 1,500 confirmed cases. But in Colombia, a recent study of nearly 12,000 pregnant women infected with Zika found zero microcephaly cases. If Zika is to blame for microcephaly, where are the missing cases?”
FOUR: It makes far more sense to listen to what South American doctors are saying about the areas where birth defects are occurring. These would be doctors who actually care about what is destroying lives and the lives that are being destroyed.
We have such reports passed along to us, thanks to Claire Robinson of GM Watch. She is one of those people who still makes the profession of journalism mean something.
Here are quotes from her most recent article, “Argentine and Brazilian doctors name larvicide as potential cause of microcephaly.”
“A report from the Argentine doctors’ organisation, Physicians in the Crop-Sprayed Towns, challenges the theory that the Zika virus epidemic in Brazil is the cause of the increase in the birth defect microcephaly among newborns.”
“The increase in this birth defect, in which the baby is born with an abnormally small head and often has brain damage, was quickly linked to the Zika virus by the Brazilian Ministry of Health. However, according to the Physicians in the Crop-Sprayed Towns, the Ministry failed to recognise that in the area where most sick people live, a chemical larvicide [pesticide] that produces malformations in mosquitoes was introduced into the drinking water supply in 2014. This poison, Pyriproxyfen, is used in a State-controlled programme aimed at eradicating disease-carrying mosquitoes.” [Emphasis added]
“The Physicians added that the Pyriproxyfen is manufactured by Sumitomo Chemical, a Japanese ‘strategic partner’ of Monsanto. Pyriproxyfen is a growth inhibitor of mosquito larvae, which alters the development process from larva to pupa to adult, thus generating malformations in developing mosquitoes and killing or disabling them. It acts as an insect juvenile hormone or juvenoid, and has the effect of inhibiting the development of adult insect characteristics (for example, wings and mature external genitalia) and reproductive development. It is an endocrine disruptor and is teratogenic (causes birth defects).”
“The Argentine Physicians commented: ‘Malformations detected in thousands of children from pregnant women living in areas where the Brazilian state added Pyriproxyfen to drinking water are not a coincidence, even though the Ministry of Health places a direct blame on the Zika virus for this damage.’”
“They also noted that Zika has traditionally been held to be a relatively benign disease that has never before been associated with birth defects, even in areas where it infects 75% of the population.”
“…The Argentine Physicians’ report…concurs with the findings of a separate report on the Zika outbreak by the Brazilian doctors’ and public health researchers’ organisation, Abrasco.”
“Abrasco also names Pyriproxyfen as a likely cause of the microcephaly. It condemns the strategy of chemical control of Zika-carrying mosquitoes, which it says is contaminating the environment as well as people and is not decreasing the numbers of mosquitoes. Abrasco suggests that this strategy is in fact driven by the commercial interests of the chemical industry, which it says is deeply integrated into the Latin American ministries of health, as well as the World Health Organization and the Pan American Health Organisation.”
“Abrasco names the British GM insect company Oxitec as part of the corporate lobby that is distorting the facts about Zika to suit its own profit-making agenda. Oxitec sells GM mosquitoes engineered for sterility and markets them as a disease-combatting product – a strategy condemned by the Argentine Physicians as ‘a total failure, except for the company supplying mosquitoes’.”
“…Abrasco added that the disease [microcephaly, other birth defects] is closely linked to environmental degradation: floods caused by logging and the massive use of herbicides on (GM) herbicide-tolerant soy crops – in short, ‘the impacts of extractive industries’.”
FIVE: In a recent greenmedinfo article—“What is the Zika Virus Epidemic Covering Up?” by Jagannath Chatterjee—the author traces other Gates-Brazil connections. For example:
“While investigating the procedures directed at pregnant women in the year 2015, shocking facts emerged.
Acting as per a WHO [World Health Organization] decision to inject pregnant women with vaccines despite contraindications the Brazilian Government had allowed its pregnant women to become the equivalent of guinea pigs.
Besides the tetanus vaccines (provided as Diphtheria Tetanus vaccines), the women had also received the Measles Mumps Rubella (MMR) vaccine in pregnancy. What is worse a DTaP vaccine was mandated for pregnant women in 2014. Citing a shortage of the DTaP vaccine the highly reactive [dangerous] DTP vaccine was also administered. Clearly huge risks had been inflicted on the unsuspecting women. None of these vaccines are known to be safe during pregnancy and the MMR and the DaPT/DPT vaccines are lapses that cannot be condoned. The rubella virus in the MMR vaccine and the pertussis component in the DPT vaccine are known to cause microcephaly…”
“The DTaP vaccine initiative to vaccinate pregnant women was financed by BMGF [Bill and Melinda Gates Foundation] funds…”
SIX: For example, every year in the US, there are 25,000 cases of microcephaly. And the literature is very clear about causes: any insult to the fetal brain during pregnancy can result in microcephaly. Severe malnutrition, falling down stairs, a blow to the stomach, a toxic street drug or medical drug or vaccine or pesticide, and so on.
SEVEN: For science bloggers who live in mommy’s basement and love the statements of the experts, try this. I’ll give you the full citation. Ready?
“Practice Parameter: Evaluation of the child with microcephaly (an evidence-based review)”; Neurology 2009 Sep 15; 73(11) 887-897; Report of the Quality Standards Subcommittee of the American Academy of Neurology and the Practice Committee of the Child Neurology Society.
Here’s the money quote:
“Microcephaly may result from any insult that disturbs early brain growth…Annually, approximately 25,000 infants in the United States will be diagnosed with microcephaly…”
Bang.
Let me take apart that quote. Microcephaly can result from any early insult to the brain. Any.
That could mean a highly toxic pesticide, for example. It could mean severe and prolonged malnutrition of the mother. It could mean a toxic substance injected into the mother—a street drug or a vaccine. It could mean a physical blow. It could mean a mother’s chronic high fever. And so on.
Moving on: 25,000 cases, not just once, but every year in the US, means what? Christopher Columbus actually brought the Zika virus to America in 1492, and it lay dormant for a very long time and then, in the modern age, exploded on the scene in the US?
No. 25,000 cases a year in the US means we’re being treated to an unsupported major bullshit story right now about the Zika virus.
That’s what it means.
EIGHT: Now we have a January 27, 2016, Associated Press story out of Rio, published in SFGate: “270 of 4,180 suspected microcephaly cases confirmed.” That’s called a clue, in case you’re wondering. Of the previously touted 4,180 cases of microcephaly in Brazil, the actual number of confirmed cases so far is, well, only 270. Bang.
But wait, there’s more. AP: “Brazilian officials said the babies with the defect [microcephaly] and their mothers are being tested to see if they had been infected. Six of the 270 confirmed microcephaly cases were found to have the [Zika] virus.”
Bang, bang, bang. Out of all the microcephaly cases re-examined in Brazil, only six have the Zika virus. That constitutes zero proof that Zika has anything to do with microcephaly.
—end of my excerpts from 2016—
Getting the picture?
In 2015-16, the World Health Organization and the press whiffed on the Zika virus-microcephaly hustle.
But they re-grouped, analyzed their mistakes, and prepared a wall-to-wall messaging campaign for the next fake pandemic.
China would provide the model:
LOCKDOWNS.
House arrest of a major percentage of the global population. Economic devastation.
COVID.
As I’ve been demonstrating for the past year, the COVID story is as full of holes as Zika.
To quote Robert W Malone again:
Ebola vaccine project for NewLink/Bioprotection Systems (rVSVdG ZEBOV Ebola vaccine project), resulting in well over 100M USD non-dilutive capital to NL/BPS. This also included working with the World Health Organization as well as initial set up of the licensing deal to Merck Vaccines of the Ebola vaccine.
I subscibe to John Rappoport and learned plenty about HIV, AIDS, Swine Flu really being from toxins sprayed on pig slurry that affected the workers and covered up by the industry. Of course, more research done to learn usually some human intervention being the cause, not some alleged virus. Malone seems to have his fingers in many pies, for sure.
Was Zika caused by Bill Gates' GMO mosquitoes?