THIS IS NOT SPIKE PROTEIN

THIS IS GRAPHENE

So, I just read in Epoch Times:

EXCLUSIVE: Why Spike Protein Causes Abnormal Blood Clots, 200 Symptoms

In this two-part paper, we aim to give an overview on COVID-19 related abnormal blood clots, how they form, how to detect them early, and how they're being treated

With all due respect, I disagree.

What doctors don't know is that poisoning with graphene oxide (and other such nanotechnology), will induce severe oxidative stress.

Severe oxidative stress will lead to destruction of the cell's DNA, apoptosis, i.e., cell death, followed by a cascade of other complications - i.e., thrombosis, strokes, heart attacks, organ failure, etc.

https://pubs.acs.org/doi/10.1021/nn201092p

Thrombus inducing property of atomically thin graphene oxide sheets

Abstract

Graphene oxide (GO), the new two-dimensional carbon nanomaterial, is extensively investigated for potential biomedical applications. Thus, it is pertinent to critically evaluate its untoward effects on physiology of tissue systems including blood platelets, the cells responsible for maintenance of hemostasis and thrombus formation. Here we report for the first time that atomically thin GO sheets elicited strong aggregatory response in platelets through activation of Src kinases and release of calcium from intracellular stores. Compounding this, intravenous administration of GO was found to induce extensive pulmonary thromboembolism in mice. Prothrombotic character of GO was dependent on surface charge distribution as reduced GO (RGO) was significantly less effective in aggregating platelets. Our findings raise a concern on putative biomedical applications of GO in the form of diagnostic and therapeutic tools where its prothrombotic property should be carefully investigated.

https://particleandfibretoxicology.biomedcentral.com/articles/10.1186/s12989-016-0168-y

Inflammatory response Graphene-Family Nanomaterials can cause a significant inflammatory response including inflammatory cell infiltration, pulmonary edema and granuloma formation at high doses via intratracheally instillation or intravenous administration.

GO PARTICLES REPORTEDLY CAUSE MORPHOLOGICAL CHANGES AND SIGNIFICANT LYSIS, LEADING TO HIGH HAEMOLYSIS IN RED BLOOD CELLS (RBCS). RBC MEMBRANE DISRUPTION IS PROBABLY ATTRIBUTED TO THE STRONG ELECTROSTATIC INTERACTIONS BETWEEN THE NEGATIVELY CHARGED OXYGEN GROUPS ON THE GO/GS SURFACE AND POSITIVELY CHARGED PHOSPHATIDYLCHOLINE LIPIDS ON THE RBC OUTER MEMBRANE.

Numerous results have shown that graphene materials cause dose dependent toxicity in animals and cells, such as liver and kidney injury, lung granuloma formation, decreased cell viability and cell apoptosis.

However, the pregnant mice had abortions at all doses, and most pregnant mice died when the high dose of reduced graphene oxide (rGO) was injected during late gestation. Notably, the development of offspring in the high dosage group was delayed during the lactation period. The high dose of GO decreased the maternal mice’s water consumption by oral exposure, which reduced milk production and thus postponed the growth of offspring. Though the findings indicate that GFNs are potentially harmful to development, but data on reproductive and developmental toxicity are still deficient.

Graphene Oxide adhered to and was wrapped in the chorion of the zebrafish embryos, causing remarkable hypoxia and hatching delay.

Graphene Oxide reportedly disrupted the alveolar-capillary barrier, allowing inflammatory cells to infiltrate into the lungs and stimulate the release of pro-inflammatory cytokines. Fibrosis and inflammation could be verified by the increased levels of the protein markers collagen1, Gr1, CD68 and CD11b in the lungs.

A high dose of Graphene Oxide that forms aggregations can block pulmonary blood vessels and result in dyspnea, and platelet thrombi were observed at high concentrations of 1 and 2 mg/kg body weight via intravenous injection.

Graphene Oxide can result in acute inflammation response and chronic injury by interfering with the normal physiological functions of important organs.

Same for amyloidosis:

https://www.sciencedirect.com/science/article/abs/pii/S0013935118306935?via%3Dihub

How graphene affects the misfolding of human prion protein: A combined experimental and molecular dynamics simulation study

Abstract

As the broad application of graphene in the biomedical field, it is urgent and important to evaluate how the graphene affects the structure and function of the proteins in our body, especially the amyloid-related proteins. Prion protein, as a typical amyloid protein, it misfolding and aggregation will lead to serious prion diseases. To explore if graphene promotes or inhibits the formation of amyloid, here, we combined the experimental and molecular dynamics (MD) simulation methods to study the influence of graphene on the globular domain of prion protein (PrP117–231). The results from fluorescence quenching and circular dichroism spectrum showed that the addition of graphene changed the secondary structure of prion protein largely, mainly reflecting in the reduced α-helix structure and the increased coil structure, indicating graphene may strengthen the misfolding inclination of prion. To further uncover the mechanism of conformational change of prion under the induction of graphene, the all-atoms MD simulations in explicit solvent were performed. Our simulations suggest that prion protein can be quickly and tightly adsorbed onto graphene together with the weak conformational rearrangement and may reorient when approaching the surface. The Van der Waals' force drive the adsorption process. In the induction of graphene, H1 and S2-H2 loop regions of prion become unstable and prion begins to misfold partially. Our work shows that graphene can induce the misfolding of prion protein and may cause the potential risk to biosystems.

Our work shows that graphene can induce the misfolding of prion protein and may cause the potential risk to biosystems