PROBLEM AND SOLUTION

According to my research on NAC, etc.

This is another conversation I'd like to share with you; something I've wanted to write for a long time....

Very briefly, because we could go on for many different studies, but I just want to capture the essence.

So, we had this exchange:

• A single dose of Sinovac gave me a stroke, respiratory arrest and a blood clot in my leg. And a nosebleed.

  Would not recommend.

• yep either transitory (like inflation) stroke or your body slowly fill with calamari

• (Me): How do you combat calamari?

  By dissolving them in N-acetylcysteine.

  I can scientifically prove it

So, here it is:

Basically, there are two factors that I see in connection with “Covid” and C-19 injections: Presence of oxidative stress and amyloid plaque formation.

In my opinion, based on research, Covid is not a "virus" - it is a misdiagnosed disease, or a set of symptoms caused NOT by a virus, but by toxicity of used substance/s, especially graphene. For reference:

https://outraged.substack.com/p/can-toxic-substances-be-mandated

https://outraged.substack.com/p/graphenenanotechnology-in-masks-and

Oxidative stress being the real cause of the so-called Covid complications and deaths is killing or damaging these people - especially those most at risk (adding to the mistreatment as the disease is misdiagnosed and thus treated with toxic drugs such as Remdesivir, rather than addressing oxidative stress with antioxidants).

The occurrence of this oxidative stress is one of the main mechanisms of graphene/nanotechnology toxicity.

https://particleandfibretoxicology.biomedcentral.com/articles/10.1186/s12989-016-0168-y “ We also point out that various factors determine the toxicity of GFNs including the lateral size, surface structure, functionalization, charge, impurities, aggregations, and corona effect ect. In addition, several typical mechanisms underlying GFN toxicity have been revealed, for instance, physical destruction, oxidative stress, DNA damage, inflammatory response, apoptosis, autophagy, and necrosis. In these mechanisms, (toll-like receptors-) TLR-, transforming growth factor β- (TGF-β-) and tumor necrosis factor-alpha (TNF-α) dependent-pathways are involved in the signalling pathway network, and oxidative stress plays a crucial role in these pathways. In this review, we summarize the available information on regulating factors and the mechanisms of GFNs toxicity, and propose some challenges and suggestions for further investigations of GFNs, with the aim of completing the toxicology mechanisms, and providing suggestions to improve the biological safety of GFNs and facilitate their wide application.”

“ROS production leading to oxidative stress

Oxidative stress arises when increasing levels of ROS overwhelm the activity of antioxidant enzymes, including catalase, SOD, or glutathione peroxidase (GSH-PX) [174]. ROS act as second messengers in many intracellular signalling cascades and lead to cellular macromolecular damage, such as membrane lipid breakdown, DNA fragmentation, protein denaturation and mitochondrial dysfunction, which greatly influence cell metabolism and signalling [175–177]. The interactions of GO with cells can lead to excessive ROS generation, which is the first step in the mechanisms of carcinogenesis, ageing, and mutagenesis [83, 122]. Oxidative stress had a significant role in GO-induced acute lung injury [30], and the inflammatory responses caused by oxidative stress often emerged upon exposure to GFNs [133, 177, 178]. The activity of SOD and GSH-PX decreased after exposed to GO in a time- and dosage-dependent manner [82, 106, 119]. Similarly, oxidative stress was the key cause of apoptosis and DNA damage after HLF cells were exposed to GO [148].”

https://www.hindawi.com/journals/bmri/2021/5518999/

https://www.nature.com/articles/am20137

The treatment that should follow because of the toxicity of the substances used - (nanotechnology is used in masks, flu "vaccines", PCR "tests", chemtrails, foods, different drugs and of course and most importantly in C-19 injections), are antioxidants. There should be prevention and treatment of oxidative stress. Oxidative stress is the reason why people have low saturation, and as a result of severe oxidative stress, cells are destroyed, apoptosis occurs, that is, cell death and a further cascade of events:

clotting, organ failure, heart attacks, strokes, etc.

The virus seems to be pretty secondary to graphene toxicity. This is nanotechnology that crosses all blood barriers, is toxic, causes oxidative stress and injures/kills people.

This is also demonstrated here:

https://expose-news.com/2022/06/27/deadly-virus-bioweapon-or-damp-squib/

So, according to these studies on graphene toxicity, Covid is not a virus, but yes, people, poisoned by nanotechnology, because of free and oxy radicals present, are susceptible to infections or flu, and these infections, as additional oxidative stress - and strain on their already struggling organisms - can be fatal to them.

When Pfizer was asked how/where and for how long "Spike Protein" is produced, it admitted they had no idea. Spike Protein seems to be just a cover for injecting people and creating a fear of pandemics. This doesn't mean that a protein or even Spike Protein can't also be used in these vaccines, but if it is used, its functions seem to have nothing to do with creating antibodies to the "virus".

But it may cause formation of misfolded proteins, AMYLOIDOSIS.

Thus, Covid thus seems to be a misdiagnosed disease. It seems to be first and foremost graphene toxicity poisoning with all that come from this toxicity such as physical destruction, oxidative stress, DNA damage, inflammatory response, apoptosis, autophagy, and necrosis. Those are causing blood clots, organ failure, strokes, and many other injuries, including death. Graphene can result in acute inflammation response and chronic injury by interfering with the normal physiological functions of important organs. Studies regarding risks of graphene in the brain show that graphene application leads to harmful effects on brain tissue development and the atypical ultrastructure was observed in the brain. Graphene demonstrates its toxicity in the central nervous system and toxicity in reproduction and development system. In the animal studies the pregnant mice had abortions at all doses, and most pregnant mice died when the high dose was injected during late gestation and the development of offspring was delayed during the lactation period. The high dose reduces milk production and postpones the growth of offspring. The developmental toxicity of graphene induces structural abnormalities, growth retardation, behavioral and functional abnormalities, and even death. Graphene induces the lung injury with inflammatory cell infiltration, pulmonary edema and granuloma formation in the lungs. Graphene causes cytotoxic effects and mitochondrial injury, leads to inflammations, induces DNA damage, decreases cell adhesion and induces cell apoptosis - cell death. Graphene inserts between the base pairs of double-stranded DNA and disturb the flow of genetic information at the molecular level, which is the main cause of its mutagenic effect. It is hemotoxic, cytotoxic, cardiotoxic, neurotoxic, harmful to reproductive system. Graphene sharpened edges cause physical destruction...

There are other toxic ingredients and heavy metals in those vials, that we already know about, such as cobalt (Co), iron (Fe), chromium (Cr), titanium (Ti)), rare earth metals such as cerium (Ce) and gadolinium (Gd), barium (Ba), cesium (Cs), aluminum (Al), silicon (Si), sulfur (S), potassium (K) and calcium (Ca) have already been found by different researchers.

https://www.aerzte-fuer-aufklaerung.de/wp-content/uploads/2022/07/Sammlung_erster_Ergebnisse_der_AG_Impfstoffe_Aufklärung_20220706.pdf

https://www.theepochtimes.com/toxic-components-found-in-blood-of-patients-who-took-covid-vaccines-german-scientists_4689152.html?utm_source=healthnoe&src_src=healthnoe&utm_campaign=health-2022-09-27&src_cmp=health-2022-09-27&utm_medium=email&est=NvKC68gYllos8ovaNYygOjIcRUDNeUDcEfhowYcx2PBrMsUVxtkSomcXWbqXA5oXdpQ4tAoZBQ%3D%3D

There is also toxic PEG! All of them will result in oxidative stress or acute oxidative stress.

https://pubmed.ncbi.nlm.nih.gov/30641367/ How graphene affects the misfolding of human prion protein: A combined experimental and molecular dynamics simulation study "Our simulations suggest that prion protein can be quickly and tightly adsorbed onto graphene together with the weak conformational rearrangement and may reorient when approaching the surface. The Van der Waals' force drive the adsorption process. In the induction of graphene, H1 and S2-H2 loop regions of prion become unstable and prion begins to misfold partially. Our work shows that graphene can induce the misfolding of prion protein and may cause the potential risk to biosystems."

So, for treatment - Glutathione precursors seem to be the most important because Glutathione is the master of all other antioxidants.

NAC helps dissolve amyloid plaque formations and blood clots and is one of the Glutathione precursors.

But in addition to this, one can take other antioxidants as well.

Regarding a potential treatment:

To address both oxidative stress and amyloid plaque formation, this is what would be helpful: Antioxidants!!! NAC, Zinc - and its ionophores, such, as Ivermectin, if available, Quercetin, Vitamin C-and others: Q10, K2MK7, Vitamin E, Resveratrol, Vitamin D, Beta Carotene, Nigella Sativa, Turmeric, etc.

https://www.hindawi.com/journals/neuroscience/2019/7547382/ - https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6500609/pdf/NEUROSCIENCE2019-7547382.pdf

5. Conclusion

ICV colchicine causes intraneuronal BAP expressions and cognitive loss which is associated with gliosis. The antioxidant NAC has reversed the cognitive deficits and inhibited microglia activation but failed to inhibit expression of BAP positive neurons and reactive astrocytes in an animal model of AD. It can be postulated that NAC might have reversed the effect of intraneuronal beta amyloid protein by acting on some downstream compensatory mechanism which needs to be explored.

https://nationaladdictionnews.com/2021/04/04/study-shows-how-the-nutritional-supplement-nac-can-help-prevent-strokes/

CORONAVIRUS, COVID-19, COVID-19 VACCINE

Study Shows How the Nutritional Supplement, NAC, Can Help Prevent Strokes

STORY AT-A-GLANCE

N-acetylcysteine (NAC) may prevent strokes in people with hereditary cystatin C amyloid angiopathy (HCCAA), a rare genetic disorder

People with HCCAA have an average life expectancy of just 30 years, and most die within five years of their first stroke

NAC appears to work by preventing the formation of amyloid-producing proteins, which promote amyloid deposits linked to strokes

The finding is even more significant because it was conducted by researchers from Children’s Hospital of Philadelphia (CHOP), which is notoriously against most dietary supplements

https://www.chop.edu/news/chop-researchers-find-supplement-prevents-strokes-patients-rare-genetic-disorder

The study by CHOP researchers suggests NAC may block the precipitation of amyloid plaque deposits, as well as help break up their formation, which could make a dramatic difference for those living with HCCAA. The study’s lead author, Dr. Hakon Hakonarson, director of CHOP’s Center for Applied Genomics, said in a news release:10

“Amyloids cannot precipitate without aggregating, so if we can prevent that aggregation with a drug [NAC] that is already available, then we could make an incredible difference in the lives of these patients.

Additionally, since we already have genetic testing available to identify these patients, we could conceivably give this treatment early in life and potentially prevent that first stroke from ever occurring.”

In the Pfizer Document - Postmarketing Experience, different Amyloidosis as an adverse result of vaccination is mentioned as well - including Cardiac Amyloidosis and Renal, Skin Amyloidosis - etc.

https://www.ahajournals.org/doi/10.1161/CIRCULATIONAHA.117.027290 Potent Thrombolytic Effect of N-Acetylcysteine on Arterial Thrombi - Results:

We demonstrated that intravenous NAC administration promotes lysis of arterial thrombi that are resistant to conventional approaches such as recombinant tissue-type plasminogen activator, direct thrombin inhibitors, and antiplatelet treatments. Through in vitro and in vivo experiments, we provide evidence that the molecular target underlying the thrombolytic effects of NAC is principally the VWF that cross-link platelets in arterial thrombi. Coadministration of NAC and a nonpeptidic GpIIb/IIIa inhibitor further improved its thrombolytic efficacy, essentially by accelerating thrombus dissolution and preventing rethrombosis. Thus, in a new large-vessel thromboembolic stroke model in mice, this cotreatment significantly improved ischemic lesion size and neurological outcome. It is important to note that NAC did not worsen hemorrhagic stroke outcome, suggesting that it exerts thrombolytic effects without significantly impairing normal hemostasis.

Conclusions:

We provide evidence that NAC is an effective and safe alternative to currently available antithrombotic agents to restore vessel patency after arterial occlusion.

This is important - lists many antioxidants and Myeloperoxidase biodegrades graphene https://www.frontiersin.org/articles/10.3389/fphys.2020.00433/full - Medicinal Herbal Compounds With the MPO-Inhibiting Activity Showing Antioxidant, Anti-Inflammation, and Neuroprotective Effects

Here is the information what markers can be done to find out if oxidative stress is present: A limitation of our study, apart from the small sample size however, is that we were unable to do laboratory testing in our patients, including checking oxidative stress markers (lipid peroxides) as well as inflammatory markers (CRP, ferritin, D-dimer) and LDH which might demonstrate a change post GSH administration [[76], [77], [78]]. A randomized, controlled trial of GSH, glutathione precursors with inflammatory/oxidative stress markers should be done in the future to fully elucidate the effects of blocking NF-kappaB, and to determine the effect of GSH and antioxidants on the clinical course of COVID-19 pneumonia and ARDS. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7172740/ - Efficacy of glutathione therapy in relieving dyspnea associated with COVID-19 pneumonia: A report of 2 cases

This is also summarized in my other post:

https://outraged.substack.com/p/short-summary

https://outraged.substack.com/p/this-is-not-spike-protein