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Blaise's avatar

Dr. Brya Ardis, MD, also reveals more filth behind Veklury® (remdesivir), in this Principia Scientific article, where he notes Veklury “is a nucleotide analogue RNA polymerase inhibitor. Dr. Ardis reveals that the symptoms of lungs filling with fluid and the other alleged COVID-19 symptoms were actually side effects of kidney poisoning and other organ damage that are known side-effects of Veklury® (remdesivir). Dr. Ardis alleges that the devestating health toll allegedly caused by COVID-19 was actually caused by the NIH recommended treatment of Veklury® (remdesivir). Dr. Bryan states that the NIH even cited two studies on its website that showed that Veklury® (remdesivir) was ineffective and unsafe to patients. It seems that many doctors just blindly followed the recommendation of the NIH to use Veklury® (remdesivir) without actually reading the cited studies.” Dr. Ardis actually tracked down those studies and read them, and a complete list of adverse effects is listed in his article here. Or you can read yourself at the New England Journal of Medicine here. Don’t have time to read it? Ok, here, let me help:

And conflict of interest (follow the money they told us at Watergate)? The following is a list of those people on the NIH Panel on COVID-19 Treatment Guidelines who had financial ties to Gilead Sciences, the manufacturer of Veklury® (remdesivir):

Rajesh Gandhi is on the advisory board of Gilead Sciences.

David Glidden is a consultant for Gilead Sciences.

Adaora Adimora is a consultant for Gilead Sciences and received research support from Gilead Sciences.

Eric Daar is a consultant for Gilead Sciences and recieves research support from Gilead Sciences.

Judith Aberg received research support from Gilead Sciences.

Jason Baker received research support from Gilead Sciences.

Susanna Naggie received research support from Gilead Sciences.

Pablo Tebas received research support from Gilead Sciences.

Roger Bedimo received an honoraria from Gilead Sciences

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Roman S Shapoval's avatar

People are so afraid of free radicals and oxidative stress, but this is why sunlight is so important - it creates free radicals in an acute way, then quells them with subcellular melatonin:

https://romanshapoval.substack.com/p/how-to-treat-emf-radiation-part-1

Love the compilation of info Mr. Human. Thank you.

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Lawdog's avatar

Remember, the FDA works for and protects Big Phama, like Gilead, and not the American people.

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Proton Magic's avatar

Very thorough examination, only there is no virus to use this drug for so that NO DRUG should be used for something never proven to exist.

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OUTRAGED HUMAN's avatar

I agree with you 100 percent. This from start to finish is nanotechnology poisoning.

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5088662/pdf/12989_2016_Article_168.pdf

GFNs were investigated to deposit in the lungs and accumulate to a high level, which retained for more than 3 months in the lungs with slow clearing after intratracheal instillation [49].

Blood-air barrier

The lungs are a potential entrance for graphene nanoparticles into the human body through airway. The inhaled GO nanosheets can destroy the ultrastructure and biophysical properties of pulmonary surfactant (PS) film, which is the first line of host defense, and emerge

their potential toxicity [54]. The agglomerated or dispersed particles deposit on the inner alveolar surface within the alveoli and then be engulfed by alveolar macrophages (AMs) [55]. Clearance in the lungs is facilitated by the mucociliary escalator, AMs, or epithelial layer [56–58]. However, some small, inhaled nanoparticles infiltrate the intact lung epithelial barrier and can then transiently enter the alveolar epithelium or the interstitium [59, 60]. Intratracheally instilled graphene can redistribute to the liver and spleen by passing through the air-blood barrier [61]. The study of blood air barrier may draw an intensive attention, since the researchers and workers occupational exposure of GFNs usually through inhalation. To make clear how the

blood-air barrier plays a role in the toxicity of GFNs may become a research hot topic.

The different administration routes influence the distribution of GFNs, for example, intratracheally instilled FLG passing through the air-blood barrier mainly accumulated and was retained in the lungs, with 47 % remaining after 4 weeks [61]. Intravenously administered

GO entered the body through blood circulation and was highly retained in the lung, liver, spleen and bone marrow, and inflammatory cell infiltration, granuloma formation and pulmonary edema were observed in the lungs of mice after intravenous injection of 10 mg kg/

body weight GO [49].

For instance, Zhang et al. found that GO was mainly entrapped in mouse lungs [49]; however, Li et al. observed that GO accumulated in mouse liver [76]. Notably, small GO sheets, with diameters of 10–30 nm, were mainly distributed in the liver and spleen, whereas larger GO sheets (10–800 nm) mainly accumulated in the lungs [49, 52, 77]. If the size of GO is larger than the size of the vessels, GO usually becomes stuck in the arteries and capillaries in the proximity of the injection site. The accumulation of GO in the lungs was shown to increase with an increase in the injected dose and size, but that in the liver significantly decreased [78].

Toxicity in internal organs GO can result in acute inflammation response and chronic injury by interfering with the normal physiological functions of important organs [32, 81]. Oral gavage experiments did not show detectable absorption of GO through the gastrointestinal tract [95]. Interesting, a low dose of GO caused serious damage to the gastrointestinal tract after maternal mice drank a GO suspension rather than a high-dose of GO because a low dose of GO without agglomeration can easily attach to the gastrointestinal surface and cause destruction through its abundant sharp edges [53]. GFNs caused inflammation and remained in the lung on day 90 after a single intratracheal instillation, and even translocated to lung lymph nodes by a nose-only inhalation [96, 97]. A high dose of GO that forms aggregations can block pulmonary blood vessels and result in dyspnea [50, 98], and platelet thrombi were observed at high concentrations of 1 and 2 mg/kg body weight via intravenous injection [89]. GO reportedly disrupted the alveolar capillary barrier, allowing inflammatory cells to infiltrate into the lungs and stimulate the release of proinflammatory cytokines [99]. Fibrosis and inflammation could be verified by the increased levels of the protein markers collagen1, Gr1, CD68 and CD11b in

the lungs. The use of Tween 80 to disperse FLG or a pluronic surfactant to disperse graphene was suggested to reduce the likelihood of lung fibrosis formation in cells or mice, whereas lung fibrosis was observed when graphene was suspended with bovine serum albumin (BSA) [100]. In addition, radioactive isotopes can be delivered into the lungs, accompanied by a depth distribution of 125I-NGO in the lungs, and the isotopes might deposit there and result in mutations and cancers [30].

In conclusion, the lung injury induced by GFNs has been studied in several studies, the results of which have demonstrated inflammatory cell infiltration, pulmonary edema and granuloma formation in the lungs.

Apoptosis

Apoptosis is defined as the self-destruction of a cell regulated by genes through complicated programmes [83, 195]. GO and rGO caused apoptosis and inflammation in mice lungs after inhalation [99], and GFNs also had pro-apoptotic effects in cells [111, 113, 124, 196].

Additionally, graphene and GO physically damaged cell membranes [166], increased the permeabilization of the outer mitochondrial membrane and changed the mitochondrial membrane potential; the increased ROS triggered the MAPK and TGF-β signalling pathways

and activated caspase-3 via mitochondrial-dependent apoptotic cascades, prompting the execution of apoptosis [83, 99]. Similarly, rGO caused apoptosis at a low dose and an early time point, triggered by the death-receptor and canonical mitochondrial pathway [110].

Blood-air barrier

The lungs are a potential entrance for graphene nanoparticles into the human body through airway. The inhaled GO nanosheets can destroy the ultrastructure and biophysical properties of pulmonary surfactant (PS) film, which is the first line of host defense, and emerge their potential toxicity [54]. The agglomerated or dispersed particles deposit on the inner alveolar surface within the alveoli and then be engulfed by alveolar macrophages (AMs) [55].

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5468375/ Graphene and the Immune System: A Romance of Many Dimensions

...

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Proton Magic's avatar

Thanks for the info. My comment was on remdesivir being used for the fake Covid 19. your saying nanotech is in Remdesivir and that's why it's toxic? Don't really follow.

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OUTRAGED HUMAN's avatar

These studies show what happens to the lungs, organs, the immune system, how nanotechnology penetrates the blood or air barrier. Nanotechnology poisoning has the same symptoms as Covid.

And nanotechnology has been applied so, what's the conclusion?

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OUTRAGED HUMAN's avatar

It is not a virus. It is nanotechnology/graphene poisoning.

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Proton Magic's avatar

Sure I understand that of course. But your post is about remdesivir and my comment was that there is no virus so no one needs remdesivir, regardless of the safety profile of remdesivir which is what your article is about. And no one should get remdesivir for graphene poisoning either.

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OUTRAGED HUMAN's avatar

there is no contradiction

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kerrylyn's avatar

Re. its sole alleged benefit - faster discharge from hospital - they forgot to add "in a coffin".

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matt. j.a.o.b's avatar

Beautiful. Thanks for the chuckle.

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jacquelyn sauriol's avatar

I would argue that Remdesiver works fabulously if you are trying to do a stealth genocide. It's perfect.

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Markker's avatar

It's already been given a nickname - Run, death is near!

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Daniel Robinette's avatar

While in the Martinsburg VA hospital they put me on the remdesivir protocol. I downloaded the African trials and proved to my doctor that I had an 89% probability of mortality because I had a viral load...covid pneumonia based on that trial. If they continued administering I would die from either liver heart or kidney failure. My doctor refused to stop. Went in with no blood clots... they did a capstan. After 6 days I had multiple pulminary embolisms... over 8 in my lungs and put me on blood thinners. My last day there they said my liver was 273 points above failure and gave me 24 hours to live if I didn't allow them to scan and fix it. I refused and told them they were pharmaceutically trained to administer drugs not heal. My wife and daughters broke me out of the hospital. My wife fed me vitamins and pounds of calfs liver for 6 days. On the 8th day my blood work showed 35 on my liver. Perfect They called me the miracle man. Even the AMA administering guide shows nurses not to Co administer hydroxychloroquine with remdesivir because it nullifies it. Remdesiver has no half life but you can't take hydroxychloroquine to nullify it with any blood thinners. So I stopped taking the blood thinners and took bromelain and tumeric 4 times a day with aspirin and took the hydroxychloroquine. In 6 months all embolisms gone.

Oh. I sent letters to the director of the hospital outlining the bonuses for administering remdesivir intravenously and the amounts madicare paid them to kill veterans. He responded that they did what they thought was right and he stood by his staff. There were 7 other veterans in the ICU with me. All believed in their country, their government and sacrificed their lives to protect it. All 7 stayed and died. Learn this lesson well. If you don't... they will kill you.

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OUTRAGED HUMAN's avatar

Of course. Oxidative stress must be addressed, that's why your protocol worked!

bromelain and tumeric - antioxidants

hydroxychloroquine - Zinc ionophore and Zinc prevents oxidative stress

With more than 6,000 scientific studies on the toxicity of graphene and its applications, including biological applications, today the mechanism of graphene toxicity is well known.

This toxicity causes DNA damage, inflammatory response, apoptosis, autophagy and necrosis, and leads to oxidative stress. Acute oxidative stress than leads to blood clots, organ failure, strokes and many other injuries, including death.

That's why addressing OXIDATING STRESS is key in "Covid" and the injuries caused by these BioNTech (bio nano technology) injections.

Also, "long Covid" is oxidative stress and must be seriously addressed before acute oxidative stress causes fatal damage!

https://outraged.substack.com/p/causes-of-injuries-and-deaths-from

https://outraged.substack.com/p/is-it-a-virus-or-is-it-graphene-toxicity

https://outraged.substack.com/p/common-denominator-for-adverse-effects

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